2011年11月15日 星期二

CATH_Angio-Seal VIP

Angio-seal VIP 的官網(內有示範vedio)


Size有兩種:6F and 8F
3 steps: Locate → Set → Seal



血管內止血器 Angio-seal 是由美國FDA獲准使用的裝置。該裝置通過一個小固定物和一塊膠原蛋白海綿(止血棉)將動脈切口夾在中間,來形成一個封閉的狀態。這些封閉物質都可以被人體吸收,並會在 60-90 天內溶解。血管穿刺後血管內止血器的使用可以將股動脈的穿刺口封閉,促進傷口的癒合,讓患者能夠更早開始行走並儘早出院。

◎術後五分鐘,床頭可抬高 30-45 度。
◎針對診斷病患術後 20 分鐘即可移動行走,一、兩個小時後便能出院。
◎針對PCI病患,可於術後 2-4 小時下床走動

ECHO_Aortic stenosis


寄件者 Echo-AS

寄件者 Echo-AS

寄件者 Echo-AS

寄件者 Echo-AS

寄件者 Echo-AS

寄件者 Echo-AS








NHLBI classification of coronary dissection









這裡有type A to C的影片

2011年1月4日 星期二

rt-PA for acute stroke

75 y/o man, Dx: Acute stroke, BW: 60Kg,於黃金3小時內到急診
和family討論後決定施打rt-PA (no contracindication)

根據台灣腦中風協會建議:
建議劑量為每公斤體重0.9毫克(最大劑量為90毫克)輸注 (infusion)60分鐘。總劑量的10%為起始劑量,以靜脈注射 (IV bolus)投與。在症狀出現後的3小時內,應儘速開始治療。

藥物: Actilyse (50mg/50ml) inj.
60*0.9=54 mg
取Actilyse 1 bt in NS 50ml成100ml,
(1) IV bolus 10ml
(2) IV pump 設定 90ml/hr
(3) 入ICU觀察

2010年11月9日 星期二

BOOKS

About ECG:
1. The ECG in acute MI_Smith
2. Challenging ECGs_Marriott
3. .....too many.......

PPM:
1.Cardiac Pacemakers Step by Step: An Illustrated Guide

EP:
1. Handbook of Cardiac Electrophysiology: A Practical Guide to Invasive EP Studies and Catheter Ablation

ECHO:
1. Textbook of Clinical Echocardiography_Otto
2. Essential Echocardiography: A Practical Handbook with DVD

Hemodynamics:
1. Textbook of Clinical Hemodynamics_Ragosta

Cardiac CATH:
1. The Cardiac Catheterization Handbook_Kern

Bible:
#### 1. Braunwald’s Heart Disease_!!!!!! Absolute bile!
2.Hurst's the Heart, 12th Edition

2010年10月19日 星期二

[CATH]acute coronary thrombus aspiration cathter

1. Fetch

Device Common Name: Embolectomy Catheter
Device Trade Name: FETCH (TM) Aspiration Catheter
http://www.medrad.com/en-us/info/products/Pages/FetchAspirationCatheter.aspx
The FETCH Aspiration Catheter is a rapid exchange catheter approved for the removal of fresh, soft emboli and thrombi from vessels in the arterial system and coronary vasculature

The Fetch Aspiration Catheter provides the outstanding handling characteristics of an AngioJet catheter with the smallest crossing profile of any manual aspiration device. The Fetch is designed for enhanced trackability and performance for small, discrete thrombus volume cases.


2. Thrombuster


3. QXT

2010年10月12日 星期二

[ECG] Parasystole

http://en.wikipedia.org/wiki/Parasystole

Parasystole
From Wikipedia, the free encyclopedia
Parasystole
Classification and external resources
MeSH D017574
Parasystole is a kind of arrhythmia caused by the presence and function of a secondary pacemaker in the heart, which works in parallel with the SA node. Parasystolic pacemakers are protected from depolarization by the SA node by some kind of entrance block. This block can be complete or incomplete.
Parasystolic pacemakers can exist in both the atrium or the ventricle. Atrial parasystolia are characterized by narrow QRS complexes
Two forms of ventricular parasystole have been described in the literature, fixed parasystole and modulated parasystole. Fixed ventricular parasystole occurs when an ectopic pacemaker is protected by entrance block, and thus its activity is completely independent from the sinus pacemaker activity. Hence, the ectopic pacemaker is expected to fire at a fixed rate. Therefore, on ECG, the coupling intervals of the manifest ectopic beats will wander through the basic cycle of the sinus rhythm. Accordingly, the traditional electrocardiographic criteria used to recognize the fixed form of parasystole are: i) the presence of variable coupling intervals of the manifest ectopic beats; ii) inter-ectopic intervals that are simple multiples of a common denominator; iii) fusion beats.
According to the modulated parasystole hypothesis, Rigid constancy of a pacemaker might be expected if the entrance block were complete, but if there is an escape route available for the emergence of ectopic activity, then clearly there must be an effective ionic communication, not complete insulation, between the two tissues. If there is an electrical communication between the two, then the depolarization of the surrounding ventricle may influence the ectopic pacemaker. That influence will be electrotonic; depolarization of the surrounding field will induce a partial depolarization of the pacemaker cells. Therefore, appropriate diagnosis of modulated parasystole relies upon the construction of a “phase response curve” as theoretical evidence of modulation of the ectopic pacemaker cycle length by the electrotonic activity generated by the sinus discharges across the area of protection. In this case, the timing of the arrival of the electronic stimulus will serve to delay or advance the subsequent pacemaker activation. In this case, the coupling intervals between the manifest ectopic and sinus discharges will be either fixed or variable, depending on the cycle length relations between the two pacemakers.


http://www.wikidoc.org/index.php/Ventricular_Parasystole

http://library.med.utah.edu/kw/ecg/ecg_outline/Lesson5/ventricular.html

Ventricular Parasystole

Non-fixed coupled PVCs where the inter-ectopic intervals (i.e., timing between PVCs) are some multiple (i.e., 1x, 2x, 3x, . . . etc.) of the basic rate of the parasystolic focus

PVCs have uniform morphology unless fusion beats occur

Usually entrance block is present around the ectopic focus, which means that the primary rhythm (e.g., sinus rhythm) is unable to enter the ectopic site and reset its timing.

May also see exit block; i.e., the output from the ectopic site may occasionally be blocked (i.e., no PVC when one is expected).

Fusion beats are common when ectopic site fires while ventricles are already being activated from primary pacemaker